| Papers [1-15] of 58 :: [Page 1 of 4] | | Go to page : 1 2 3 4 —> | Search results on "MYOCARDIAL INFARCTION PULMONARY EDEMA": |
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Myocardial Infarction and Pulmonary Edema, 2008.
An case study assessment and diagnosis of a patient with anteroseptal myocardial infarction (MI) and pulmonary edema.
2,445 words (approx. 9.8 pages), 9 sources, APA, $ 74.95
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Abstract
The paper summarizes a patient's condition of anteroseptal myocardial infarction (MI) and pulmonary edema. The paper discusses the medications currently being administered to the patient and details the more pertinent drugs. The paper also outlines the short and long-term care of the patient, particularly in terms of nursing management. The paper then explains that this patient has a life-threatening chronic illness and concludes that future complications as well as another MI will only be avoided by strict compliance with health teaching about medications, diet and lifestyle.
Outline:
Introduction
Assessment and Medical Diagnosis
Pathophysiology
Medications
Nursing Interventions
Conclusion
From the Paper
"The medical diagnosis for Patient 00-065 is anterial MI and pulmonary edema. Myocardial infarction is defined by Fenton and Stahmer (2006, p. 1) as "the rapid development of myocardial necrosis caused by a critical imbalance between the oxygen supply and demand of the myocardium". The cause usually is thrombus formation in a coronary vessel. To assess the patient and to identify and categorize the MI that can range from unstable angina to ST-elevation MI, doctors use cardiac markers. ST-elevation and other categories will be identified from the electrocardiogram."
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Myocardial Infarction Vs Angina, 2008.
This paper compares the assessment of myocardial infarction versus angina.
707 words (approx. 2.8 pages), 4 sources, APA, $ 25.95
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Abstract
This paper relates that evaluation of chest pain is possibly one of the most important elements of nursing assessment skills. The paper then provides an assessment of myocardial infarction versus angina and shows how discerning angina from a myocardial infarction is a difficult task, requiring the rapid use of evaluation skills as well as the prompt administration of treatment.
Outline:
Introduction
Clinical Paper
Conclusions
From the Paper
"Chest pain is notoriously difficult to assess. Depending on the signs and symptoms, findings on the ECG and lab results the management of the patient differs significantly. The classic presenting symptoms of a myocardial infarction (MI) is chest pain or discomfort. Angina pectoris may present the same way but will generally be of shorter duration. Both may be described as pain, pressure, tightness, heaviness, burning or squeezing. Both may radiate into arms, shoulders, jaw or back. Not all patients will have these classic symptoms."
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Myocardial Infarction, 2004.
This paper discusses the occurrence of a myocardial infarction.
675 words (approx. 2.7 pages), 2 sources, MLA, $ 23.95
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Abstract
This paper looks at the myocardial infarction (MI), or in other words a type of heart attack. The writer discusses what happens physically and physiologically when an MI occurs. The writer also examines the risk factors for MI. In this article, the writer studies the treatment and prognosis for MI.
From the Paper
"Myocardial infarction (MI) is the name for a heart attack, which occurs when there is a sudden and complete blockage of the flow of blood to a section of the heart muscle. Myocardial MI's can occur at anytime and they occur without warning. With age, the coronary arteries become narrowed because of a build up of plaque along the walls leading to a condition known as arteriosclerosis. This means the blood flow through the vessels is lowered and the blood supply to the muscle of the heart ... "
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Thrombolysis for Myocardial Infarction in Accident and Emergency, 2004.
A look at national standards for emergency cardiac care for patients entering the accident and emergency system.
5,690 words (approx. 22.8 pages), 53 sources, MLA, $ 137.95
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Abstract
This paper examines the standards set by the National Service Framework for coronary heart disease regarding emergency cardiac care for patients presenting to the accident and emergency system at a hospital. The paper explains that these standards place a priority on aggressive management and providing early thrombolysis and that this type of treatment is controversial because it is questionable whether, given the suggested timeline and the need to differentiate from non-cardiac causes of chest pain, an adequate history and physical can be performed to prevent the administration of thrombolytics in patients where they are contraindicated. To further examine this topic, the paper gives a clear definition of acute myocardial infarction, looks at how to diagnose a patient with acute chest pain, explores the history, indications, and contraindications of thrombolysis, and reviews a multidisciplinary approach to thrombolytic administration.
From the Paper
"The definitive diagnosis of AMI is best obtained by following a standard chest pain protocol. Most accident and emergency wards have these in place. It is standard to initially obtain a 12 lead electrocardiogram (ECG) and begin cardiac monitoring. Patient?s routine laboratory studies include electrolytes, blood urea nitrogen (BUN), complete blood count (CBC) and markers for myocardial injury (Creatinine Kinase isoenzyme-myocardial (CK-MB) or troponin). Normal serial CK values rule out an acute infarction but are negative in the setting of acute unstable angina. A slight rise in CK-MB or troponin indicates myocardial injury but is not specific for ischemic syndromes. Troponin assay is highly sensitive for identifying acute coronary syndromes. Troponin has longer half life in the system than CK-MB but is less specific for the identification of infarction as opposed to repeated episodes of myocardial ischemia. As the total CK greater than two times the upper ranges of normal is indicative of infarction it can be used as an adjunct in diagnosis ischemia versus infarction and also in determining the relative efficacy of reperfusion. Serum troponin may take up to six hours to become diagnostically sensitive enough (Dougan, 2001)."
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Myocardial Infarction, 2005.
An overview of the causes and treatments of heart attacks.
1,150 words (approx. 4.6 pages), 8 sources, APA, $ 39.95
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Abstract
This paper describes heart attacks, also known as myocardial infarctions. The paper begins by discussing how a person experiences a heart attack, including symptoms of an MI. Next the paper lists the risk factors for heart attacks, such as gender differences. The paper concludes with a discussion of the importance of perceiving certain symptoms as a threat to health.
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Noncardiogenic Pulmonary Edema and Heroin Overdoses, 2005.
A proposal to study noncardiogenic pulmonary edema in heroin overdoses among patients in the Washington DC Metro area.
2,816 words (approx. 11.3 pages), 9 sources, APA, $ 83.95
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Abstract
This paper proposes a study is to determine if heroin users in the Washington DC metropolitan area who overdose and use antihistamines are less likely to develop NCPE than those who overdose and do not use antihistamines. The study uses a predictive correlational design. The independent variable is antihistamine use and the dependant variable is development of NCPE. The study is to take place in two Washington DC metropolitan hospitals and the subjects are heroin users without any pre-existing pulmonary condition other than asthma, admitted to the hospital in the past five years and diagnosed as having a heroin overdose.
Outline:
Abstract
Introduction
Problem/Research Question
Purpose
Hypothesis
Definition of Terms
Limitations
Conceptual Framework
Review of Literature
Design and Setting
Population and Sample
Instrument
Validity and Reliability
Data Collection Procedure
Data Analysis Procedures
From the Paper
"Following a heroin overdose, a person may develop one, or several different reactions to the drug. Pulmonary edema can occur as the result of increased permeability of the capillaries in the lungs. The lungs swell and fill with fluid, and if this condition is left untreated it can lead to death. Histamine is thought to increase capillary permeability, thus furthering the edema process. If antihistamines lower the histamine level, and decrease the chances of patients who are taking them to develop pulmonary edema after a heroin overdose, then these patients need to be cared for differently than those patients who are not taking antihistamines. Patients who are taking antihistamines at the time of their overdose would not need to have x-rays taken to see if edema has developed, thus avoiding needless exposure to radiation. "
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Edema, 2004.
An insight into the cause, symptoms, and treatment methods for pulmonary edema and peripheral edema.
1,127 words (approx. 4.5 pages), 5 sources, MLA, $ 39.95
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Abstract
This paper examines the condition of edema, a medical condition that results from an increase in accumulation of water and sodium inside the body. It looks at how there are many different types and equally different contributing factors that cause edema and focuses on two common types, namely, pulmonary edema and peripheral edema. It also discusses how edema is a symptomatic response of an underlying disorder that needs to be identified and treated and how it is important to understand that the use of diuretics is administered as a palliative rather than curative treatment.
Outline
Introduction
Edema in General
Pathophysiology of Edema
Pulmonary Edema
Peripheral Edema
Conclusion
From the Paper
"Two factors are essential for the formation of edema. First is a change in the fluid exchange system of the body. This is bought about by a shift in the pressure gradient in between the interstitial space and the plasma. Secondly increased levels of sodium bought about by its retention by the kidney. The presence of sodium prevents the uniform distribution of water, which is mainly retained in the extra cellular fluid. Osmolarity of sodium is the main reason for this non-uniform distribution of the excess water. The presence of excess water in the extra cellular fluid can trigger abnormal reactions in the organs leading to impairment of normal functions."
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The Pathophysiology of Edema, 2004.
This paper discusses the cause, symptoms, and treatment methods for two common types of edema: pulmonary edema and peripheral edema.
1,150 words (approx. 4.6 pages), 6 sources, MLA, $ 39.95
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Abstract
This paper explains that there are two causes of edema: a change in the fluid exchange system of the body, and an increased level of sodium bought about by its retention by the kidney. The paper points out that, for treating swollen legs, it is best to begin with simple exercises and postures (leg raise); in most cases of non-systemic peripheral edema, these simple treatment methods will reverse the condition. The paper stresses that, unless it is an emergency, the use of diuretics should be minimized, as it may result in complications resulting from over-diuresis.
Table of Contents
Introduction
Edema in General
Pathophysiology of Edema
Pulmonary Edema
Treatment
Peripheral Edema
Conclusion
From the Paper
"Pulmonary edema also known as ?Lung water? or ?Pulmonary congestion? is a condition where fluid accumulates inside the lungs leading to respiratory difficulties. The chief cause for this problem is the decreased activity of the heart resulting in increased pressure on the pulmonary veins. As a result, fluid is forced into the alveoli and this directly interferes with the gaseous exchange mechanism leading to shortness of breath."
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Primary Pulmonary Hypertension, 2002.
An overview of the pulmonary vascular disease primary pulmonary hypertension (PPH) including causes and symptoms.
1,150 words (approx. 4.6 pages), 11 sources, $ 44.95
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Abstract
This discusses primary pulmonary hypertension (PPH). It is a pulmonary vascular disease characterized by an elevation in mean pulmonary artery pressure and pulmonary vascular resistance. Recently, PPH gained national attention because of its association with appetite suppressants. PPH may also be associated with pregnancy, hypothyroidism, autoimmune disorders, human immunodeficiency virus infection, and the use of drugs such as oral contraceptives and cocaine. Patients with PPH may report dyspnea on exertion and fatigue. Early diagnosis is crucial.
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Pulmonary Embolism and Air Travel, 2006.
Comparing information from web sites on pulmonary embolism.
1,145 words (approx. 4.6 pages), 4 sources, MLA, $ 39.95
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Abstract
This paper is written in the first person and compares information found on three sites on the Internet about pulmonary embolism. The first website is more oriented at the seasonal effects of long flights and pulmonary embolism. The second website is a news website with an article about pulmonary embolism and air travel. The third website stresses the need for knowing more about pulmonary embolism and the environment in the airplane. The author draws conclusions and recommendations from the websites and checks with the New English Journal of Medicine's detailed study about the distance traveled by air and the number of patients who developed the condition to confirm the findings.
From the Paper
"My son and daughter-in-law had made plans for France. They landed at the airport and their daughter started to experience chest pains. The emergency unit at the airport checked her and told them something about it probably being pulmonary embolism. They took her to the hospital and my daughter-in-law called me to ask me about pulmonary embolism. I checked the search engines hoping to get directed to authentic information on the net about this. I came across three websites that seemed authentic and derived information from there and compared them."
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Chronic Obstructive Pulmonary Disease, 2008.
A look at the causes, treatment, management and prevention of chronic obstructive pulmonary disease (COPD).
2,007 words (approx. 8.0 pages), 5 sources, MLA, $ 63.95
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Abstract
This paper discusses how obstructive pulmonary disease (COPD) is the fourth leading cause of death in the United States and worldwide even though it is not a contagious disease and how there is no know cure for COPD. It also looks at how COPD is a disease of the lungs caused by damage to the airways primarily due to cigarette smoking and how prolonged exposure to dust, chemicals and pollution also cause COPD. In addition, the paper describes the symptoms, causes, treatment, management and prevention of the disease. Furthermore, the paper emphasizes that if an individual smokes, the first and most critically important step in addressing chronic obstructive pulmonary disease (COPD is to quit smoking and to do so immediately.
Outline:
Introduction
COPD: Symptoms
Treatment of COPD
Goals of Treatment in COPD
COPD Medications
Components of the Pulmonary Rehab Program
Prevention of Progression of COPD
Management and Prevention of Problems
Summary and Conclusion
From the Paper
"As stated in the previous section there are treatments for COPD and according to the National Heart, Lung and Blood Institute "quitting smoking is the single most important thing" that the individual can do in order to reduce the risk of the development of 'chronic obstructive pulmonary disease (COPD) and to slow the progression of the disease COPD. The goals of treatment for COPD are: (1) to relieve the symptoms with no to little side experience of side effects; (2) to slow the disease progression; (3) to improve the individual's tolerance for exercise; (4) prevention and treatment of complications or onset of problems occurring suddenly; and (5) to improve the individuals' health overall. (National Heart, Lung, and Blood Institute: Disease and Conditions Index, 2007) It is important to note that the National Heart, Lung, and Blood Institute relates that treatment of COPD is "different for each person" and that the individuals doctor might well refer the individual to a lung specialists or 'pulmonologist'. (2007) "
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Pulmonary Hypertension, 2002.
An insight into the cause and treatment of primary and secondary pulmonary hypertension.
1,150 words (approx. 4.6 pages), 9 sources, $ 44.95
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Abstract
This paper discusses primary and secondary pulmonary hypertension. It includes a description of pulmonary hypertension and the differences between primary and secondary pulmonary hypertension, including the cause and course of the disease followed by symptoms and treatment.
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Chronic Obstructive Pulmonary Disease (COPD), 2008.
This paper explores emergency department admission for chronic obstructive pulmonary disease (COPD).
3,222 words (approx. 12.9 pages), 20 sources, APA, $ 92.95
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Abstract
The paper describes chronic obstructive pulmonary disease, including its diagnosis, current treatments and its interdisciplinary care. The paper examines the guidelines and implementation of the 2007 "Australian and New Zealand Guidelines for the Management of Chronic Obstructive Pulmonary Disease", also known as the COPD-X Plan, and focuses on emergency nursing roles and practices. The paper limits itself to looking at aspects of diagnosis and treatments of acute exacerbation of COPD. The paper also compares the COPD-X Plan with the "Global Initiative for Chronic Obstructive Lung Disease" guidelines. The paper concludes that while there is a need for more research to derive more empirically-established and consistent guidelines for interdisciplinary use, present guidelines in use must be adhered to as the next best measure for COPD treatment until such evidence is present.
Outline:
Introduction
Literature Review
Discussion/Critique
Conclusion
From the Paper
"Chronic obstructive pulmonary disease (COPD) has remained one of the top ten diseases resulting in death worldwide. It is the third and second most prevalent and burdensome disease in Australia and New Zealand, respectively (McKenzie, Frith, Burdon & Town, 2007).It is also among the top ten causes of mortality in Australia and New Zealand, the fourth in the United States and been noted to be rising (McKenzie, Frith, Burdon & Town, 2007; Smithline, Rowe, Radeos, Cydulka & Camargo, 2005). In spite of these facts, there have been inconsistencies between the goal of reducing morbidities and mortalities associated with COPD and the means by which the different health policy societies have outlined. These inconsistencies are manifested in the construction of recommendations of different national and international societies such as the American Thoracic Society, European Respiratory Society and British Thoracic Society - recommendations which, for all intents and purposes, should be uniform and unambiguous (Ferguson, 2000). This especially pertains to aspects of diagnosis and treatment. While these discrepancies may seem trivial, their cumulative impact can be seen above."
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Pulmonary Embolism, 2005.
This paper examines a pulmonary embolism, an infection that can often lead to fatal problems.
1,125 words (approx. 4.5 pages), 4 sources, $ 44.95
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Abstract
The discussion identifies the symptoms of a pulmonary embolism, the various forms of treatment that are available and the possible interventions of physicians in order to preserve a patient's life. The paper explains that due to the time sensitive nature of a pulmonary embolism, it is likely that many diagnoses will be fatal if not treated appropriately and explains that therefore these issues must be taken seriously at all times.
From the Paper
"In the world of illness and disease, various forms of infection often lead to significant problems that may be fatal for some patients. One such case is pulmonary embolism, which is typically caused by other underlying circumstances that often create a dangerous situation for patients suffering from this condition. It is necessary to identify the specific criteria that are related to this condition, which include the following: pathophysiology of the condition, tools for diagnosis, evaluation processes for consideration, possible interventions for implementation and issues related to this diagnosis in current practice. In a diagnosis of pulmonary embolism, a number of symptoms are considered, including chest pain, abdominal pain and acute respiratory problems (Feied and Handler Para. 20)."
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Chronic Obstructive Pulmonary Lung Disease, 2007.
A discussion of chronic obstructive pulmonary lung disease (COPD).
1,178 words (approx. 4.7 pages), 5 sources, MLA, $ 40.95
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Abstract
The paper offers a definition and description of chronic obstructive pulmonary lung disease (COPD). The paper looks at the causes, symptoms, conventional diagnosis, transmission, prognosis and prevention of the disease. The paper seeks to highlight how COPD has long-term and frequently deadly consequences for those who refuse to engage in a healthy lifestyle.
From the Paper
"The online MedlinePlus medical encyclopedia defines Chronic Obstructive Pulmonary (Lung) Disease (also known as COPD) as a group of lung diseases which cause swelling of the airways; the same source then lists Emphysema and "chronic bronchitis" as two of the most common forms of COPD (sec.3). To expand upon the last sentence a little more fully, the National (U.S.) Heart, Lung, and Blood Institute defines Chronic Obstructive Pulmonary Lung Disease as being, fundamentally, a progressive state of airway constriction and obstruction that ultimately leads to dramatically reduced lung function - even fatally reduced lung function (1-2). To summarize, Chromic Obstructive Pulmonary Disease may best be described as an incurable and progressive disease that attacks the pulmonary capacity of the lungs; in particular, the disease either (in the case of chronic bronchitis) causes mucus build-up in the bronchial tubes or (in the case of emphysema) sufficiently irritates the alveoli that they become "stiff" and unable to hold air - thereby ensuring that the body is not taking enough oxygen into it at the same time as it cannot get rid of unwanted carbon dioxide (American Academy of Family Physicians, para.1-4)."
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