| Papers [1-4] of 4 | Search results on "APOLICIA AREATA": |
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Apolicia Areata, 2002.
An overview of the hair loss disease, alopicia areata.
900 words (approx. 3.6 pages), 3 sources, $ 35.95
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Abstract
This paper will discuss the cause and affects of alopicia aAreata, and discover how hair loss is part of way of life. By learning how the disease is formed, we can what is becomes in the stages of the cause. By learning some the ways that is enhanced, we can see the different dimensions of the disease beings formed. This is the way the disease will presented in this essay.
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Alopecia Areata, 2007.
This paper discusses alopecia areata, a disease resulting in partial or complete hair loss.
2,879 words (approx. 11.5 pages), 10 sources, MLA, $ 85.95
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Abstract
The paper discusses the nature and causes of alopecia areata. The paper explains that some show a strong link between alopecia areata and genetics, while others suggest that environmental factors may contribute to this condition. This paper reviews the general epidemiology of alopecia areata, the contributing factors and the relationship of animal models of experimentation and study to finding new treatments for the disease. The paper relates that the future of research will continue to focus on identifying risk factors for developing alopecia areata, including possible environmental triggers and genetic factors increasing one's susceptibility to the condition.
Outline:
Overview
Epidemiology: Alopecia Areata
Animal Models and Alopecia Areata
Conclusion
From the Paper
"Based on the research that is available most researchers consider alopecia areata an autoimmune disease, suggesting that the body's own immune system, which normally protects the body, attacks the hair and hair follicles resulting in hair loss or an immune reaction. Still others have proposed certain viruses may contribute to the disease, though there is presently little evidence supporting this claim. Most at risk for alopecia areata include those with close member of their family with the disease. As more is learned and uncovered about this complex condition, researchers are likely to uncover other risk factors for alopecia areata."
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Alopecia, 2005.
A discussion of alopecia areata, a hair loss disorder.
2,304 words (approx. 9.2 pages), 10 sources, MLA, $ 71.95
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Abstract
Alopecia areata is a systemic hair loss disorder, which affects roughly around 4.7 million people in the United States alone. The paper explains that it is characterized as an autoimmune disease that leads to either localized or complete hair loss. The effects of the disease may either be permanent or reversible depending on the nature and extent of damage to the hair follicles. It is believed that both genetic as well as environmental factors have an influence in the onset of the condition. This paper provides a brief overview of the different types of alopecia and discusses in detail the possible pathophysiology and the treatment options for the disease.
Introduction
Alopecia (Different Types)
Hair Follicles (Complex Morphognesis)
Androgenic Alopecia
Molecular Pathology of AGA
Immunology of Alopecia Areata
Microcirculation in Scalp Tissues
Treatment for Alopecia (Cortisones and Anti Inflammatory and Immunotherapy Drugs)
Cosmetic Surgery
Conclusion
From the Paper
"Alopecia is a disease triggered by the immune response of the body, which attacks and alters the normal development cycle of the hair follicles. Normally each hair follicle is in any one of the following three stages of life cycle namely anagen (growth phase), catagen (regression phase) and telogen (resting phase). Alopecia areata refers to the significant hair loss observed due to the immune response against the hair follicles in the anagen stage, resulting in severe reduction or complete stoppage of the hair growth phase. When the hair loss is observed throughout the body it is called as alopecia universalis. When complete hair loss is confined to the head, the condition is referred to as Alopecia totalis while hair loss that is localized and observed as patchy bald regions is known as Alopecia areata. [Ralph Paus]"
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Alopecia, 2006.
This paper examines the causes and effects of alopecia areata, a systemic hair loss disorder which affects around 4.7 million people in the United States alone.
2,141 words (approx. 8.6 pages), 10 sources, MLA, $ 67.95
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Click here to show/hide summary
Abstract
This paper describes alopecia areata, a systematic hair loss disorder, as a disease that is independent of race, gender or age, hence affected people represent a wide and diverse group. The writer details the effects of the disease which can be either permanent or reversible depending on the nature and extent of damage to the hair follicles. It is believed that both genetic and environmental factors have an influence in the onset of the condition. While there is no complete cure as of yet for alopecia, treatment is primarily aimed at slowing down and containing the severity of the symptoms. This paper also discusses the psychological devastation suffered by the affected person which at times can be debilitating. This paper contains a brief yet concise overview of the various types of alopecia as well as the possible treatment options available.
Table of Contents:
Introduction
Alopecia (Different Types)
Hair Follicles (Complex Morphognesis)
Androgenic Alopecia
Molecular Pathology of AGA
Immunology of Alopecia Areata
Microcirculation in Scalp Tissues
Cosmetic Surgery
Conclusion
Bibliography
From the Paper
"Research into the pathophysiology of AGA though still inconclusive, has offered enough insight and has identified the important role of androgens in the progressive miniaturization of the hair follicles leading to baldness. Testosterone and its metabolite dihydrotestosterone (DHT) in particular have been directly related as causative factors. The presence of the enzyme 5[Alpha]-reductase is essential for the catalytic reduction of testosterone into the more potent form of DHT. Chromosome 2p has the gene necessary for encoding 5[Alpha]-reductase. Extensive studies conducted on people suffering from AGA have revealed an excess of 5[Alpha]-reductase, and consequently greater amounts of DHT. DHT affects the hair follicles by binding with the dermal papilla cells creating androgen receptor complexes. Androgen receptors act as transcription factors and are directly involved in the synthesis of the protein substances that affect the hair follicles."
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