Abstract
This paper examines the novel mechanism of disease transmittal in prions as well as the diseases themselves (mad cow disease and Creutzfeld-Jacobs disease). Functions of normal prions are also discussed. The paper also examines how prions are devoid of nucleic acids, and thus carry no information other than their tertiary structure.

From the Paper
"Knockout mice survived up to their 70th week, and then they began to display signs common with brain disease, which included a loss of motor coordination. Curiously, the symptoms shown by these knockout mice were similar to those found in many cases of prion diseases."

Tags:brain, cow, creutzfeld, disease, infectious, jacobs, mad, prions, protein