An analysis of the evidence that stuttering is related to brain damage.
Written in 2008; 1,298 words; 4 sources; MLA; $ 43.95
Paper Summary:
This paper relates that hypotheses that attempt to explain stuttering point to the roots of this condition in the neurophysiology and/or neuropsychology of the individual experiencing the dysfluency. The paper further discusses that, while theories do exist that place stuttering into a behavior dependant on learning and conditioning due to environmental aspects, the fact remains that stuttering, whether learned or conditioned, is the result of a neurophysiological response manifested as dysfluent speech. The paper maintains that, upon examination of the nature of stuttering both in terms of research into etiology and effective treatment, the conclusion that stuttering is the result of brain damage is the one most applicable and probable.
From the Paper:
"According to earlier research statistics, approximately 25 to 35 percent of individuals who have developed stuttering sustained a form of brain damage, either at birth or during early childhood (Guitar, 2006). The reported factors, including anoxia at birth, premature birth, head injury, and mild cerebral palsy, all point to a disruption in the fetal development of a normal central nervous system (Guitar, 2006). Since there appears to be this correlation between brain damage and stuttering, research has been done to determine how brain functions are different in these individuals. Electroencephalographic (EEG) studies have shown a possible right-hemisphere dominance in terms of language production in individuals who stutter and cerebral blood flow (CBF) studies have shown these results as well (Guitar, 2006). "
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