A case study investigating if Obestatin is a ligand on GPR36 and its related GPCRs.
Written in 2008; 7,060 words; 25 sources; MLA; $ 158.95
Paper Summary:
The paper explains the pharmacology behind G-protein-coupled receptors (GPCRs) and the manner in which they function. The paper explains the seriousness of obesity and details the numerous medications that are currently in use, together with their mode of action. Part ll of the paper analyzes whether the medication Obestatin is a ligand on GPR39 and its related GPCRs. The paper is well documented with tables and graphs and provides results with the conclusion.
Outline:
General Information
Obestatin Transient Investigation
Introduction
Method
Results and Discussion
From the Paper:
"The mainstay of treatment for obesity is an energy-limited diet and increased exercise. Although adherence to this regimen can cure obesity, many patients are unable to make the required sacrifices. It leads to drug application and bariatric surgery come into play. Bariatric surgery in both risk assessment and financial expenditure excess the drug application, therefore the drug treatment is the most popular and prevalent therapy in tackling obesity disease. Currently, the most commonly prescribed medication for diet/exercise-resistant obesity is orlistat (Xenical(r) which reduces intestinal fat absorption by inhibiting pancreatic lipase) and sibutramine (Reductil(r), Meridia(r) which is an anorectic). In pursuing more efficacy and safer drugs, scientists have plunged into the researches in the past ten years and identified some of the genes responsible for the body weight and fat contents. They pointed out the associate protein products of these genes play critical roles in regulating the energy balance. "
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