This paper details the results of clinical test data which prove that by properly lowering blood cholesterol levels in patients age 65 and older they'll receive similar benefits to those of much younger adults.
Written in 2006; 1,120 words; 1 sources; APA; $ 38.95
Paper Summary:
This paper details the morbidity and mortality risks of coronary heart disease in elderly patients. The writer stresses the importance of proper treatment and deterrence which is fundamental to developing the length and worth of life for the geriatric residents at large. The results of the clinical test data in this paper have confirmed that patients age 65 and older receive similar advantage from blood cholesterol decline as younger adults. Drug therapy in this populace, while safe, necessitates cautious surveillance and dose modification due to potentially distorted drug metabolism and concomitant medications.
Table of Contents:
Introduction
Assessment and Diagnosis
Treatment
Combination Therapy
Conclusion
Resources
From the Paper:
"The cholesterol that builds up in atherosclerotic lesions starts off principally from plasma lipoproteins, predominantly low-density lipoprotein cholesterol (LDL-C). Continuous hypercholesterolemia results in optimistic cholesterol equilibrium at the vascular interface, with more lipoprotein-bound cholesterol entering than exiting the arterial wall. This directed net invasion of lipoprotein-bound cholesterol into the vascular arterial intima speeds up the development of atherosclerosis. Oxidation of LDL-C in the subendothelial layers of the arterial wall, which can be shaped by triggered endothelial cells, smooth muscle cells, or monocytes/macrophages, appears to be a key irreversible event in the atherogenic process. As an atherosclerotic lesion develops below an intact layer of endothelial cells, macrophages may discharge toxic products, including superoxide anions, cytokines, and proteolytic enzymes, which can lead to endothelial dysfunction."
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