A review of the biological and cognitive theories of depression and the evidence which supports or contradicts these theories.
Written in 2009; 1,096 words; 10 sources; APA; $ 38.95
Paper Summary:
This paper looks at the main biological theory of depression, which is known as the monoamine-deficiency hypothesis. The paper first discusses the controversy surrounding anti-depressant medications and the effect of serotonin on the brain and highlights the difficulty in proving the efficacy of these drugs. The paper then looks at cognitive theories of depression, with a focus on Aaron Beck's cognitive model, and notes the studies that support and validate this theory. The paper points out that no single theory about such a complex phenomenon as depression can be considered 'complete'.
Outline:
Biological Theories of Depression
Cognitive Theories of Depression
From the Paper:
"Research into the causes of depression since the 1950s has given rise to several 'biological' theories of depression, including perhaps the most popular hypothesis that a deficiency (or imbalance) of important neurotransmitters such as noreineprine and serotonin in the brain are largely responsible for major depressive disorder (Alladin, 2005, p.8). This biological theory of depression is also called the Monoamine-Deficiency Hypothesis. The theory emanated from the study of early antidepressants developed in the 1950s, which increased the availability of neurotransmitters (norepinephrine and serotonin) in certain areas of the brain and seemed to help alleviate major depressive disorder in a large number of patients (Belmaker and Agam, 2008)."
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