This paper discusses the pathophysiology of childhood asthma by presenting a case study of a five year-old child at the Pediatric Intensive Care Unit.
Written in 2004; 2,835 words; 9 sources; MLA; $ 84.95
Paper Summary:
This paper presents a detailed case of a child with asthmatic symptoms that are discovered to be exacerbated by Parainfluenza A, which contributed to the onset of his asthma. The author points out that the severity of asthma varies greatly from child to child, from a cough associated with exercise or at night to a catastrophic shortness of breath that leads to death. The paper points out that the inability of endogenous, circulating, catecholamines to induce reversal of bronchoconstriction may reflect one or more defects in the patient with asthma. Figure: Asthma Mechanisms
Table of Contents
CC
Pt. Introduction
Admission to System
Pt. Physical Assessment
Lab Data
Isolated for parainfluenza
Diagnostic Data
Medications-Previous
Medications Ordered
Singular
Flovent MDI
Albuterol MDI
Solumedrol
Ranitidine
Ceftriaxone
Treatment
Clinical Course
Pathophysiology of CC
Lab Data
Diagnostic Data
Medications
Summary
From the Paper:
"The primary mechanical effects of asthma on the lung as a result of BMP are increased work of breathing to overcome airway obstruction and hyperinflation attributable to progressive air trapping results from a ball-valve effect in the airways and causes ventilation/perfusion (V/Q) mismatch and right-to-left shunting of unoxygenated blood through the lungs. V/Q mismatch and the increased oxygen demand on the respiratory muscles as they work to overcome airway obstruction result in significant hypoxia early in the course of a wheezing episode. This hypoxia stimulates tachypnea, which partially compensates for the V/Q mismatch and is reflected by significant hypocarbia. If airway obstruction is not rapidly reversed, air trapping and V/Q mismatch can progress till decreased ventilation occurs, Pco2 rises, and ultimately respiratory failure ensues."
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